Tuesday 21 April 2026

Science Daily

  1. Scientists at UC Riverside have found a clever new way to outsmart termites—by turning their own instincts against them. Using a natural pine scent called pinene, which smells like food to termites, researchers can lure the pests straight toward a targeted dose of insecticide hidden in wood. The result is dramatically higher kill rates—jumping from about 70% to over 95%—without the need for widespread toxic fumigation.
  2. After two centuries of failed attempts, scientists have finally grown dolomite in the lab, cracking a long-standing geological puzzle. They discovered that the mineral’s growth stalls because of tiny defects—but in nature, those flaws get washed away over time. By mimicking this process with precise simulations and electron beam pulses, the team achieved record-breaking crystal growth. The finding could reshape how high-tech materials are made.
  3. Many of the world’s largest river deltas—home to hundreds of millions of people—are sinking faster than rising seas, according to a sweeping global study. Using high-resolution satellite radar maps, researchers found that human activities like groundwater pumping, reduced sediment flow, and rapid urban growth are driving widespread land subsidence across 40 major deltas.
  4. AI-powered personas are becoming so realistic that they can infiltrate online communities and subtly steer public opinion. Unlike traditional bots, they adapt, coordinate, and refine their messaging at a massive scale, creating a false sense of consensus. Early warning signs—like deepfakes and fake news networks—have already appeared in global elections. Researchers warn that the next election could be the true test of this technology’s power.
  5. Cancer cells are known for their “glutamine addiction,” but many can escape this weakness by switching to alternative fuels. Researchers found that vitamin B7 acts like a metabolic “license,” enabling this escape route through a key enzyme. Without biotin, cancer cells lose that flexibility and stop growing. Mutations in a cancer-linked gene can make this vulnerability even stronger, offering a promising new target for therapy.

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