Thursday 5 March 2026

Science Daily

  1. A sweeping new study of more than 2,000 insect species reveals a troubling reality: many insects may be far less capable of coping with rising temperatures than scientists once hoped. Researchers found that while some species living at higher altitudes can temporarily boost their heat tolerance, many insects in tropical lowlands—where biodiversity is highest—lack this flexibility. Because insects play essential roles as pollinators, decomposers, and predators, their vulnerability could ripple through entire ecosystems.
  2. Popular weight-loss drugs such as Ozempic, Wegovy, and Mounjaro may do more than help people shed pounds. New research suggests these GLP-1 medications could also help protect the heart after a heart attack by restoring blood flow in tiny blood vessels that often remain blocked even after doctors reopen a major artery.
  3. Scientists have uncovered a crucial weakness in the malaria parasite that could open the door to new treatments. Researchers identified a protein called Aurora-related kinase 1 (ARK1) that acts like a traffic controller during the parasite’s unusual cell division process, ensuring its genetic material is properly separated as it multiplies. When scientists switched off ARK1 in laboratory experiments, the parasite could no longer replicate correctly and failed to complete its life cycle in both humans and mosquitoes—effectively halting its ability to spread.
  4. A new ultrathin photodetector from Duke University can sense light across the entire electromagnetic spectrum and generate a signal in just 125 picoseconds, making it the fastest pyroelectric detector ever built. The breakthrough could power next-generation multispectral cameras used in medicine, agriculture, and space-based sensing.
  5. A new experimental drug is showing remarkable promise for children with Dravet syndrome, a severe genetic form of epilepsy. In clinical trials, the treatment zorevunersen cut seizures by as much as 91% while also improving quality of life for many patients. The therapy works by boosting the function of a key gene involved in nerve cell signaling. Encouraging results have led researchers to launch a larger Phase 3 trial.

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